ID

45536

Descrizione

Principal Investigator: Dmitry A. Gordenin, PhD, National Institute of Environmental Health Sciences (NIEH, DHHS), Research Triangle Park, NC, USA MeSH: Neoplasms https://www.ncbi.nlm.nih.gov/projects/gap/cgi-bin/study.cgi?study_id=phs000677 *Reprinted from Roberts et al. "An APOBEC cytidine deaminase mutagenesis pattern is widespread in human cancers", Nature Genetics, 45:970-976, 2013, with permission of Nature Publishing Group:* Recent studies indicate that a subclass of APOBEC cytidine deaminases, which convert cytosine to uracil during RNA editing and retrovirus or retrotransposon restriction, may induce mutation clusters in human tumors. We show here that throughout cancer genomes APOBEC-mediated mutagenesis is pervasive and correlates with APOBEC mRNA levels. Mutation clusters in whole-genome and exome data sets conformed to the stringent criteria indicative of an APOBEC mutation pattern. Applying these criteria to 954,247 mutations in 2,680 exomes from 14 cancer types, mostly from The Cancer Genome Atlas (TCGA), showed a significant presence of the APOBEC mutation pattern in bladder, cervical, breast, head and neck, and lung cancers, reaching 68% of all mutations in some samples. Within breast cancer, the HER2-enriched subtype was clearly enriched for tumors with the APOBEC mutation pattern, suggesting that this type of mutagenesis is functionally linked with cancer development. The APOBEC mutation pattern also extended to cancer-associated genes, implying that ubiquitous APOBEC-mediated mutagenesis is carcinogenic.

collegamento

dbGaP study = phs000677

Keywords

  1. 29/12/22 29/12/22 - Simon Heim
Titolare del copyright

Dmitry A. Gordenin, PhD, National Institute of Environmental Health Sciences (NIEH, DHHS), Research Triangle Park, NC, USA

Caricato su

29 dicembre 2022

DOI

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Licenza

Creative Commons BY 4.0

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dbGaP phs000677 An APOBEC Cytidine Deaminase Mutagenesis in Human Cancers

Subject ID, sample ID, and sample use variable obtained from participants with cancer and involved in the "An APOBEC Cytidine Deaminase Mutagenesis in Human Cancers" project.

pht003581
Descrizione

pht003581

Alias
UMLS CUI [1,1]
C3846158
Subject ID
Descrizione

SUBJECT_ID

Tipo di dati

string

Alias
UMLS CUI [1,1]
C2348585
Consent group as determined by DAC
Descrizione

CONSENT

Tipo di dati

text

Alias
UMLS CUI [1,1]
C0021430
UMLS CUI [1,2]
C0441833
Source repository where subjects originate
Descrizione

SUBJECT_SOURCE

Tipo di dati

string

Alias
UMLS CUI [1,1]
C3847505
UMLS CUI [1,2]
C0449416
UMLS CUI [1,3]
C0681850
Subject ID used in the Source Repository
Descrizione

SOURCE_SUBJECT_ID

Tipo di dati

string

Alias
UMLS CUI [1,1]
C2348585
UMLS CUI [1,2]
C3847505
UMLS CUI [1,3]
C0449416
Case control status of the subject
Descrizione

AFFECTION_STATUS

Tipo di dati

text

Alias
UMLS CUI [1,1]
C3274646

Similar models

Subject ID, sample ID, and sample use variable obtained from participants with cancer and involved in the "An APOBEC Cytidine Deaminase Mutagenesis in Human Cancers" project.

Name
genere
Description | Question | Decode (Coded Value)
Tipo di dati
Alias
Item Group
pht003581
C3846158 (UMLS CUI [1,1])
SUBJECT_ID
Item
Subject ID
string
C2348585 (UMLS CUI [1,1])
Item
Consent group as determined by DAC
text
C0021430 (UMLS CUI [1,1])
C0441833 (UMLS CUI [1,2])
Code List
Consent group as determined by DAC
CL Item
General Research Use (GRU) (1)
C0021430 (UMLS CUI [1,1])
C0242481 (UMLS CUI [1,2])
SUBJECT_SOURCE
Item
Source repository where subjects originate
string
C3847505 (UMLS CUI [1,1])
C0449416 (UMLS CUI [1,2])
C0681850 (UMLS CUI [1,3])
SOURCE_SUBJECT_ID
Item
Subject ID used in the Source Repository
string
C2348585 (UMLS CUI [1,1])
C3847505 (UMLS CUI [1,2])
C0449416 (UMLS CUI [1,3])
Item
Case control status of the subject
text
C3274646 (UMLS CUI [1,1])
Code List
Case control status of the subject
CL Item
Case (2)
C3274647 (UMLS CUI [1,1])

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